Raw research findings for this medication. Mostly single studies, not curated or confirmed robust.
Tenoxicam is a non-steroidal anti-inflammatory drug (NSAID) of the oxicam family, closely related to piroxicam. It blocks the COX enzymes that make prostaglandins and lasts long enough to be taken once a day. It is used for osteoarthritis, rheumatoid arthritis and painful inflammation of joints and tendons. It is sold as Mobiflex and Tilcotil, among other names, outside the US.
Tenoxicam is broken down mainly by the liver enzyme CYP2C9, and it already stays in the body for several days. People with reduced-function versions such as *2 and *3 clear it more slowly, so levels rise higher and stay longer. CPIC has published a guideline on CYP2C9 and the NSAIDs, including tenoxicam.
Clinical guidelines that name a gene for Tenoxicam. They sort people into groups by the gene copies they carry.
CPIC guideline: CYP2C9. CYP2C9 activity score 0 to 2. Strong. 2020.
Every published finding on gene variants and Tenoxicam. Upload your DNA file to see which of these rows apply to you.
PharmGKB weighs all studies of a variant and rates how strong the evidence is, from 1A (strongest) to 4.
CYP2C9 CYP2C9*1, CYP2C9*2, CYP2C9*3: Level 1A · replicated. Metabolism/PK. 2.
Study findings and evidence levels: PharmGKB (CC BY-SA 4.0). Guidelines: CPIC and DPWG, as bundled in PharmCAT. Most studies were done mainly in people of European ancestry.
More drugs from the same category
Not a prescribing recommendation. This page is educational information about pharmacogenomic research and clinical guidelines - it is not medical or dosing advice, and not a prediction of how any medication will work for you. Never start, stop, or change a medication or dose based on it; talk to your doctor or pharmacist, who can order clinical-grade testing if appropriate.
Content updated September 2026.
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